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Be explored in more detail.O22 Comparison of a rapid visual algorithm for quantification of infarct size with direct planimetry of infarct size by delayed enhancement-CMR Omar M Cheema, Ankit A Patel and Dipan J Shah Methodist DeBakey Heart Vascular Center, Houston, TX, USAExample of non-ischemic fibrosis. Subepicardial delayed enhancement of the anteroseptal and anterior walls (a) and inferior
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Nhanced cardiovascular magnetic resonance (CMR) has the unique ability to visualize and quantify myocardial scarring. It is well understood that myocardial infarctions resulting from transplant vasculopathy adversely affect prognosis and modify therapy. There is a growing body of evidence from non-transplant disease states, that the presence of non-infarct myocardial scar is also correlated to poo
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Holesterol in R (48 ?42) compared to Z (27 ?42) mg/dl, p = 0.07. The final total cholesterol was similar between groups. No between group changes in HDL or triglycerides were seen. See Table 1 for changes in vessel wall parameters in the 2 groups. Plaque volume regressed in R while it progressed in Z and total vessel volume followed the same trend (p = 0.11). No between group differences in lumen
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Chemic injury. Overall, patients with DE had scores ranging from 1 to 19, with a mean of 5.4 (standard deviation = 4.8). Non-ischemic DE was most commonly seen in the anterolateral and inferior walls (Figure 3). There was no significant association between the presence of DE and time since transplant or current biopsy result. Conclusion: DE is a common feature in the transplant population. Most DE
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D (HE) regions (signal intensity > 2 standard deviations above remote myocardium) in all short axis DE-CMR slices. The total volume of the HE zone was then divided by the total volume of myocardium within the left ventricle (calculated by planimetry of endocardial and epicardial borders on all DECMR images so as to include both HE and non-HE myocardium). Visual scoring of all studies was performed
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Nhanced cardiovascular magnetic resonance (CMR) has the unique ability to visualize and quantify myocardial scarring. It is well understood that myocardial infarctions resulting from transplant vasculopathy adversely affect prognosis and modify therapy. There is a growing body of evidence from non-transplant disease states, that the presence of non-infarct myocardial scar is also correlated to poo
1
Patient group.O20 Characterization of tissue heterogeneity by contrast-enhanced cardiovascular magnetic resonance imaging is a powerful predictor of ventricular tachyarrhythmias on ambulatory holter ECG in hypertrophic cardiomyopathy Caitlin J Harrigan1, A Selcuk Adabag2, Evan Appelbaum3, Kevin S Heffernan1, John R Lesser2, James E Udelson1, Warren J Manning4, Barry J Maron2 and Martin S Maron1 1
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Infarct sizes was 0?0 by direct planimetry. Bland Altman analysis revealed a nonsignificant bias of 2.1 of LV (p = NS) with 95 confidence interval between the visual algorithm and planimetry of +11.3 to -7.1 , see figure. The visual algorithm required less time than direct planimetry (5.5 ?1.6 minutes vs. 22.4 ?4.5 minutes respectively; p

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